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Is ATP free energy? – Tesla Free Energy Generator Youtube Subs

Is this the mechanism of a calorie restriction-dependent upregulation of cellular function? These are interesting but complex questions. Perhaps ATP free energy is not merely a marker of mitochondrial biogenesis (as the authors are quick to assert) but it might also be a critical energy reserve in skeletal muscle, which is a major source of energy for many of the body’s functions. Perhaps the important thing is that ATP plays a crucial role in the functioning of skeletal muscle. We need to better understand the relationship between cellular energy and mitochondrial capacity without relying too heavily on a single molecule of energy. It seems like a good idea to test this theory using a sample of animal and human subjects who are currently calorie-restricted but who are not under the care of an eating-deprivationist. I’d also like to see an estimate as to how much ATP is required for activity of all enzymes in skeletal muscle. If an estimate of the energy requirements of cellular energy metabolism is obtained, this should explain both the dramatic increase in mitochondrial biogenesis that we have observed and the apparent decline in muscle fiber number. The main limitation to this study lies in the fact that no one was able to measure energy expenditure or substrate oxidation by either the subjects or their subjects’ muscles. Nevertheless, the current study is interesting so far as it establishes that the changes in muscle fiber number observed in humans in response to calorie restriction are associated with ATP-dependent biogenesis of mitochondria in skeletal muscle.[8] However, there are still many technical problems in interpreting this paper. For instance, it is not clear whether increasing the amount of ATP taken from the blood by muscle cells will result in a net energy surplus or be an increase in oxidative phosphorylation of some of the enzymes. However, this paper is rather clear that the changes in muscle fiber number observed in response to calorie restriction can be associated with increases in substrate oxidation, with the effect of increasing the rate of ATP production. Indeed, when the subjects were only allowed one hour of recovery between bouts of exercise, it was the increase in total cellular ATP synthesis that accounted for the difference in change in muscle number.[18] However, that does not necessarily mean that the same increase, if it occurs, will have the same effect on skeletal muscle fiber number. Perhaps the changes in the protein composition of the muscle fiber, its size and number, that occur as a result of exercise, have a significant effect on the net cellular ATP production that is linked to mitochondrial biogenesis? This could be investigated further if another study is conducted using additional samples of animals

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